Hyperventilation is Defined as an Increase in Respiratory Rate and/or Tidal Volume, Resulting in the Elimination of More Carbon Dioxide than the Body Produces
Hyperventilation Results in Hypocapnia (Decreased Arterial pCO2) and Respiratory Alkalosis (at Least Initially, Until Renal Compensation Results in Bicarbonate Excretion with an Eventual Decrease in the pH Back Toward Normal) (see Respiratory Alkalosis)
Respiratory Alkalosis is Defined as an Acid-Base Disorder Characterized by a Decrease in Arterial pCO2 with an Associated Increase in Arterial pH (at Least Initially)
Note that a Patient Can Have a Respiratory Alkalosis without Being Alkalemic
Example
Due to Normal Compensatory Mechanisms, Chronic Respiratory Alkalosis Induces Metabolic (Predominantly Renal) Compensation (with a Progressive Decrease in Serum Bicarbonate Over Time), Culminating in Only Minimal/Absent Alkalemia