Atrial tachycardia is a form of supraventricular tachycardia which originates within the atria but outside of the sinus node
Atrial tachycardia does not require the atrioventricular (AV) node, accessory pathways, or ventricular tissue for initiation and maintenance
Epidemiology
Occurrence in the Structurally Normal Heart
Occurrence in the Structurally Abnormal Heart
Congenital Heart Disease: especially after surgery for repair or correction of congenital or valvular heart disease
Physiology
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Classification Method Based on Endocardial Activation
Focal Atrial Tachycardia: arises from a localized area in the atria such as the crista terminalis, pulmonary veins, coronary sinus ostium, or intra-atrial septum
Reentrant Atrial Tachycardias: reentrant atrial tachycardias most commonly occur in persons with either structural or complex heart disease, particularly after surgery involving atrial incisions or scarring
Usually macroreentrant
Classification Method Based on Pathophysiologic Mechanism
Structural Heart Disease: including ischemic, congenital, post-operative, and valvular heart disease
Iatrogenic Atrial Tachycardias: typically result from ablative procedures in the left atrium (several typical origination sites for these tachycardias have been identified, including the mitral isthmus between the left lower pulmonary vein and mitral annulus, the roof of the left atrium, and, for reentry, around the pulmonary veins)
Classification Method Based on Location of the Arrhythmogenic Focus
Right Atrial Origin
Left Atrial Origin
Diagnosis
Echocardiogram: useful to assess for structural heart disease
Chest CT: may be used to rule out pulmonary embolism, map pulmonary veins (prior to ablation), etc
P-Wave Morphology: abnormal (due to its ectopic origin) and may give clue to the site of origin and mechanism of the atrial tachycardia
Focal Tachycardia: the P wave morphology and axis depend on the location in the atrium from which the tachycardia originates
Macroreentrant Circuits: P wave morphology and axis depend on activation patterns
P-Wave Axis: usually abnormal (ie: inverted in II, III and aVF)
Isoelectric Baseline (unlike atrial flutter)
AV Block May Be Present: due either to physiologic response to the rapid atrial rate (or in cases due to digoxin intoxication, due to the vagptonic effects of digoxin)
Treatment
Rate Control with AV Nodal Blocking Agents
Adenosine (see Adenosine): used for atrial tachycardia due to triggerred activity
Diltiazem (see Diltiazem): used for atrial tachycardia due to triggerred activity
Metoprolol (see Metoprolol): used for atrial tachycardia due to triggerred activity or enhanced automaticity
Verapamil (see Verapamil): used for atrial tachycardia due to triggerred activity
Anti-Arrhythmics
Refractory Recurrent Atrial Tachycardia: Class Ic antiarrhythmics
Maintenance of Sinus Rhythm: Class III antiarrhythmics
Cardioversion: for cases where rhythm is not well-tolerated hemodynamically and/or in whom rate-control drugs are ineffective or contraindicated
Radiofrequency Catheter Ablation: for symptomatic, medically refractory patients
Surgical Ablation: for patients with complex congenital heart disease